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What you should know about Hepatitis

I have the yellow fever doctor says a patient. How do you know you have yellow fever? She asked the doctor. Patient response “Doctor, I have a fever and my eyes are yellow.”

Herein lies the sea of ​​confusion between yellow fever, jaundice and hepatitis. There seems to be much confusion about the trio of yellow fever, jaundice and hepatitis. For many people, it seems the fever with yellow eyes and mucous membranes and skin (technically called Jaundice) must be yellow fever.

However, in the science of medicine, there is a big difference between these diseases. Yellow fever virus causes yellow fever. A wide range of viruses or other toxins can cause hepatitis. Both diseases cause inflammation of the liver. Inflammation of the liver is unable to effectively degradation of red blood cells. This results in the accumulation of bilirubin in the blood and connective tissues leading to what is called jaundice. The theme of yellow fever is beyond the scope of this article. Suffice to say that is a very deadly entirely preventable disease with high mortality, but with a vaccine affordable but effective yellow fever which is taken once every ten years.

The specific viruses that cause viral hepatitis include hepatitis A (HAV); Hepatitis B virus (HBV); Hepatitis D (HDV) Delta agent; Hepatitis E virus (HEV) transmitted in epidemic form in Asia, North Africa and Mexico. Other viruses that have been implicated in causing hepatitis such as hepatitis G virus (HGV), SEN-V and TT virus (TTV) virus.

HAV causes the sporadic or epidemic causes of hepatitis. The virus is transmitted or spread by contaminated food and water sources, therefore, which can lead to outbreaks in large population groups. Its spread is favored by crowding, poor personal hygiene. The incubation period is usually a few days, 30 days on average. The disease is usually self-limiting with complete recovery within two weeks. The long duration of infection does not occur. The infection can be severe in adults, but can occur in children without any symptoms or effect of health problems. The death of hepatitis A is very rare.

Hepatitis B is usually transmitted by inoculation of infected blood or blood products or through sexual intercourse. The virus (HBV) can be found in blood, saliva, semen and vaginal fluids of infected people. An infected mother can transmit HBV to the child at the time of delivery. The risk of chronic infection in such cases is very high. HBV tends to occur frequently in homosexual or gay men and injecting drug users (IDUs). Certain groups of people are more at risk of infection; doctors, nurses, dentists, blood bank staff, patients and staff of hemodialysis centers, etc. Unfortunately, less blood is needed to transmit HBV HIV that causes AIDS.

From the time of infection, it takes 6 weeks to 6 months (on average 12-14 weeks) for the infected person to become clinically ill. Hepatitis B can also be in the way the very serious hepatitis called fulminant hepatitis in approximately 1% of those infected. In this group, death may occur in up to 60% of them. In another group, the disease can persist as chronic hepatitis B. In this group, the infection persists for years and then the patient will progress to liver cirrhosis and liver cancer.

Hepatitis D (Delta-MAIL)
Hepatitis D is a defective virus is unable to cause hepatitis on its own but in association with HBV. HDV can therefore co-infect a person with HBV or super person infected with chronic hepatitis. When it occurs simultaneously with acute hepatitis or severe chronic hepatitis leading rapidly to cirrhosis.

It can be transmitted through blood transfusions, intravenous drug use, sexual contact and from mother to child. Therefore, having multiple sexual partners may increase the risk of infection with hepatitis C. The incubation period is about 6-7 weeks.

This is one of the aqueous hepatitis virus. Outbreaks occur in India, Burma, Afghanistan, Algeria and Mexico. The disease is self-limiting with no carrier state. But mortality is usually higher in infected pregnant women infected (up to 10-20%).

This virus was detected in blood donors, injecting drugs, hemodialysis patients, hemophiliacs and patients with chronic hepatitis B or C. Hepatitis G does not appear to cause significant liver disease.

Clinical hepatitis IMAGE
The onset of illness may be sudden or insidious. Usually it is a flu-like illness with malaise, fever, muscle pain, joint pain, fatigue, sore throat, runny nose and loss of appetite, nausea and vomiting. There is often diarrhea, constipation and aversion to smoke smoking. While the fever begins to wane, jaundice sets in 5-10 days. Refrigerate or chillness may occur. The liver is enlarged and tender to the touch. The spleen and lymph node can also increase in size. The type of hepatitis diagnosis is facilitated by various laboratory tests.

The treatment of hepatitis includes bed rest, if any. The diet should consist of palatable foods identified by the patient. Fluid replacement and multivitamin supplements are treated. The drug alpha interferon has been used in patients with acute hepatitis C and found to reduce the risk of progression of chronic hepatitis C

In general, the careful handling of needles and blood tests before transfusion are useful ways to prevent hepatitis. Hepatitis A can be prevented by vaccination and immunoglobulin use in people who have close contact with patients infected with HAV. Hepatitis B is a highly effective and potent vaccine, which gives protection to life and requires booster only intermittently. Hepatitis B immunoglobulin may also protect or lessen the severity of disease if given within seven days of exposure to HBV.

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